Clinical Image | Open Access
Volume 2023 - 2 | Article ID 233 | http://dx.doi.org/10.51521/WJCRCI.2023.220126
Academic Editor: John Bose
Aaron Niblock1,2, Nyasha Chikanya1
1Haematology
department, Antrim Area Hospital, Northern Ireland
2School of Medicine, Ulster University, Northern Ireland
Corresponding Author: Aaron Niblock, Haematology department, Antrim Area Hospital, Northern Ireland; School of Medicine, Ulster University, Northern Ireland
Citation: Aaron Niblock, S Rajendran (2023). Microangiopathic Haemolytic Anaemia Induced by COVID-19. World J Case Rep Clin Imag. 2023 October-November; 2(2)1-3.
Copyrights © 2023, Aaron Niblock, et al. This article is licensed under the Creative Commons Attribution-Non-Commercial-4.0-International-License-(CCBY-NC) (https://worldjournalofcasereports.org/blogpage/copyright-policy). Usage and distribution for commercial purposes require written permission.
Introduction:
Microangiopathic Haemolytic Anaemia(MAHA) is a descriptive
term for the presence of fragmentation of red cells (schistocytes) which can be
demonstrated on a blood film [1]. It occurs when endothelial damage and/or
fibrin deposition damages red cells. The
presence of this associated with a thrombocytopenia is an emergency as the
known differential includes DIC, TTP, HUS, Malignancy, and HELLP all of which
can ultimately end life [2]. Since Covid19 pandemic we report
another case in this differential that resolves with conservative management.
Case Report:
An 84-year-old male presented to our emergency department with a 5 days’ history of unwellness. He reported lethargy, loss of appetite and a non-productive cough. Clinical examination was significant for tachycardia, clinical dehydration and bibasal crepitations. An ECG done showed that the man was in atrial fibrillation with rapid ventricular response. Subsequent investigations revealed an acute kidney injury with a significant drop in glomerular filtration from baseline, new anaemia and thrombocytopenia and he tested positive for Covid 19. Blood film was performed which showed true thrombocytopenia with schistocytes of 2.5% and subsequently on repeat had increased to 4%. A plain chest radiograph showed bilateral patchy inflammatory changes consistent with Covid 19. Kidneys were described as normal on ultrasound. The patient was subsequently treated for covid 19 with dexamethasone, received intravascular fluids for the AKI and serial monitoring of his renal function. Following treatment of covid 19 the patient improved significantly and the AKI improved and was discharged with no renal follow up.
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